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Albatross from My Father pg.2

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During the clean up, T. Gondii victims out of range for induced aggression and immune survivors were quarantined with all non-latent infected individuals released after antibiotic treatment. This reconstruction period saw food preservative reintroductions and increased plastics reliance.

Pneumonia incidents were quickly responded to out of relapse fears, but treatments slowed after T. Gondii was ruled out. The culprit, part of the Mimiviridae family, was relatively harmless. While exterminating all infected persons had been proposed due to parasites’ ability to inject their genome into hosts, early T. Gondii tests found only sterile junk RNA produced.

I don’t know if my dad was among those abused by CDC guards, but he’d always refused to go into town. I’d been living with my mother a few months before he got sick.

Containing the largest viruses, Mimiviridae is the sole family capable of being infected by smaller viruses or virophages. After the fractured T. Gondii genome reverse transcribes within the virus under the required conditions, localized mutation near instantaneously becomes violently exponential, producing the “explosive” effect for the first few minutes of infection.

Like any virus, the Human Genome Virus’s purpose is to reproduce. However, HGV does so by moving directly into the genome instead of eventually being absorbed into the genome of epidemic survivors. During infection, HGV “randomly” activates and or deactivates genes, altering cells and cell parameters within the former human self-ecosystem. In “nature”, cells burst during this phase or soon quickly die from lack of a functional genome.

Prior to the first outbreak, certain plastics were found capable of filling in DNA sequence “holes”. While pollution reduction was of priority in reconstruction, a decade of accelerated food production guaranteed at least minor water contamination. Though consumption had long been associated with gene interruption, the integration of plastic molecules through the food chain received relatively little notice.

With the global population susceptible to Mimiviridae and/or T. Gondii, early containment of the second outbreak would have been impossible had HGV maintained its ancestral transmition instead of fluid contact.

With most “pocket evolutions” being fatal, cell parameters limited any particular regional mutation within mutation’s success or failure from affecting the overall organism’s survivability as shown in cases of “limbs” separating from the main host.

During the “explosive” stage, host nervous systems degraded to the most basic evolutionary function in serving doubly as both sensory and motor systems during neural rewiring, explaining initial retardations in movement and reasoning. By the time seperated “limbs” began adherring to other hosts for information communication, collaberation, increased intelligence, and produced independently evolved titan class organisms, the military response removed their offensives as a threat.

The myth of the second outbreak as transhuman evolution, and it’s genetic applications regarding positve eugenics and exocolonization will be further pondered in chapter 5.

Of no relevance to this book, I spoke to a member of the Lizard Eaters tribe once while on vacation in Australia. He said his tribe never heard anything.